EMF Blocking and even EMF Shielding

If you reside in an apartment or a house, or you just want to keep your home free of electromagnetic fields There are plenty options to limit your exposure. One of the most effective is to reduce your use of electronic devices. You can also turn to EMF blocker paint to block EMF radiation from reaching your house. Another way to shield your house from EMF radiation is to use an RF shielding canopy. This is a cloth made of net which contains EMF shielding and is used to stop EMFs from entering a room. Another option is to have your home equipped with an electrical enclosure. These devices are called Faraday cages.

Several studies have shown how the non-ionizing energy of RF has antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to result from the deregulation the cancer stem cell. This could explain the long-term responses seen in certain patients suffering from advanced HCC. But, the reason for AM RF EMF's effect in cancer patients is not yet clear.

emf radiation blocker  of AM RF EMFs on HCC tumor growth in vivo was studied in mice.  Check out here  were divided into 3 groups. The first group was not exposed to RF EMF. Second group members were exposed to RF EMF at frequencies similar to the frequency used by humans. In the third, they were subjected to RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. The results revealed that tumours treated with HCCMF were significantly shrinking. However, the tumors treated with RCF showed no evidence of shrinkage of the tumor.

The reason for cancer-specific AM RF EMF might be based on the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is caused through CACNA1H the protein that is responsible for the influx of Ca2+ specific to tumours. The results suggest that CACNA1H could have more broader implications for the treatment and diagnosis of different cancers.

The tumours in those in the group that were unaffected to EMF from radiofrequency, and fed a normal mouse diet. The tumors of those in the HCCMF group were treated with Huh7 cells after they were five-seven weeks old. The tumors were then killed in cases of excessive burden.

The tumours in the three groups also showed distinct growth curves. The tumours treated with HCCMF saw a significant reduction in tumour size after eight weeks. However, the tumors which were treated by RCF showed no shrinkage. The difference was highly significant. The tumours treated with RCF had necrosis, which is typical in tumors that have been that are exposed to RCF. It is possible that this necrosis was due to an absence of oxygen in the more invasive tumors.

In sum, the results show an AM-RF EMF is a powerful source of anticancer activity in vitro and in vivo. Several studies have shown it is true that AM RF EMF produces measurable reduction in tumours in HCC patients. There is  emf radiation blocker  that AM RF EMF triggers these effects due to CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may cause a lasting effect on the development of HCC tumours in vivo.