If you reside in a house or apartment or simply need to ensure that your house is free from EMFs, there are a number of ways you can reduce exposure. One of the most effective is to reduce the use of electronic devices. It is also possible to use EMF block paint to prevent EMF radiation from entering your home. Another easy way to protect your house from EMF radiation is to install an RF shielding canopy. This is a cloth made of net which contains EMF shielding. It's used to stop EMFs from entering rooms. Another option is to get your home equipped with an enclosure that is conductive. These enclosures are known as Faraday cages.
Several studies have shown that the non-ionizing RF EMF has antiproliferative effects on HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed to result from the deregulation of cancer stem cells. This could explain the long-term effects observed in some patients with advanced HCC. But, Discover more for AM RF EMF's effect in patients with cancer is not yet clear.
The effects on the effects of AM RF EMFs on HCC tumor growth in vivo were studied in mice. The tumors were split into 3 groups. First, block emf that was unaffected to RF EMF. The second group was exposed to RF EMF at the same frequency to the frequency used by humans. In the third, they were subjected to RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was evaluated against that of RCF. The results indicated that tumours treated with HCCMF had significant shrinkage. However, the tumours treated with RCF showed no evidence of shrinkage in the tumour.
The reason for tumour-specific AM RF EMF could be due to the fact that cancer cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is caused by CACNA1H which is a protein which is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have wider implications for the treatment and diagnosis of different cancers.
The tumours in the controls were never exposed to EMF from radiofrequency, and fed a normal mouse diet. The tumors of the HCCMF group were treated with Huh7 cells when they were five-seven weeks old. The tumors were removed after they had a high burden.
The tumors of the three groups also displayed distinct growth curves. The tumors treated with HCCMF had a significant reduction in tumour size after 8 weeks. However, the tumors which were treated by RCF didn't show reduction in size. The difference was significant. The tumors treated with RCF had necrosis, which is typical in tumours exposed to RCF. It is possible that the necrosis is caused by a lack of oxygen in the larger tumours.
In emf blocking clothing , the findings show that AM RF EMF has anticancer activity in vitro and in the vivo. Numerous studies have demonstrated that AM RF EMF produces measurable tumour shrinkage for HCC patients. It is possible that AM RF EMF causes these effects due to CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting impact on the development of HCC tumors in vivo.